Medical Conditions - Blog Posts

📂 🙏🏾

QAQ I hope this satisfies!

Asylum bakugou? Like, when he was around 4-8 years old, every day afterschool he'd have to go see a psychologist at this asylum place, and he didn't want deku to be around him much cuz there's something wrong with him and he can't change that.

This leads to deku thinking his quirklessness is why bakugou stays away from him but that's not the case. Bakugou has intense anger issues due to a chemical imbalance in his body thanks to his quirk. He needs to go for coping skills and meditation. Or else his anger is 10x worse.

It gets better after a couple years. Now he just goes once a week, and then, the SLUDGE VILLAIN ATTACK TAKES PLACE.

Shit went down hill and he has to go for therapy 3 times a week.

Then USJ, sports festival and Kamino happen and bakugou needs to go to therapy nearly everyday, his classmates think he doesn't like hanging out with people when in reality, he can't because therapy. but it isn't enough, none of the meds are working well enough and the only way to take care of it was training training training even more because if he was better ALLMIGHT WOULD HAVE NEVER RETIRED. he works himself to exhaustion.

Eventually it gets so bad his stress is making his chemical imbalance worse and he's lashing out more and more. The provisional licence exam was fucked. But the deku vs Kacchan helped a wittle.

Aizawa pulls him to the side after a really bad 'tantrum' (God I hate this word) to scold him but all bakugou can do is shake and run to his dorm. Aizawa and the class were dumbfounded. They went to Bakugou's room to see what's wrong but they just walk in to see him wreaking his room in utter panic looking for something, a white bottle...

Aizawa stooped in and talked to him in private to figure out what's going on. The others wanted to know what's going on but decided to wait like the good angels they are. Bakugou mutters out a brief explanation. You see, since bakugou is in the dorms, he can't exactly go to therapy. He goes once every 2 weeks but in reality he needs to go 2 times a week. So the boy's been pretty fucked out. Aizawa is upset that bakugou kept it in all this time and at himself for not reaching out sooner or even looking through the student files more in detail.

Not to mention his parents only get him his meds when they're almost empty. They cant bother to get even a backup. Just when it's almost done. They cause 3/4 of his stress.

So, Aizawa calls for a staff meeting with bakugou there to get his view and opinion. Its gonna be a long night. Midnight, the bitch, made a comment how if he runs out of meds then they can tie him up like the sports festival. Cementoss and allmight chuckle (I'm passive aggressive). They didnt realize katsuki flinching and ever so slightly trembling. Dadzawa notices. He is LIVID.

Recovery girl shut them up and told in the most deadpan voice "I'm ashamed to have coworkers like you".

Recovery girl takes bakugou outside. She turns to katsuki and says "child, I'll get those medication for you one way or another, either with the school's medical finances or my own." She then went to call some people to buy the meds in bulk. While rubbing Bakugou's back which is so soothing for him that he sleeps.

Inside the room, aizawa had just finished chewing out the others. Nezu also said "that is rather inappropriate ta know?" While sipping tea like the dictator he might be. Aizawa and mic call Bakugou's parents and well, they aren't exactly liars. Cue, Dadzawa and present dad with katsuson. ❤❤❤

Deku and bakugou have a long talk after everything is taken care of (the next day). The others watch out for Bakugou's condition and even visit his therapist, Ms. Aiko. She's a god sent. Everything improves much more. Bakugou has never felt better. He feels something he hasn't for a long time. He feels normal, validated. Himself for once.

Thus, the story of bakugou getting love and comfort while still being the badass #1 he is and always will be.

I hope this satisfies. ❤

Cirrhosis Of The Liver

Introduction

Cirrhosis stands as a challenge in the narrative of liver disease, marking the culmination of a complex interplay of factors that converge to undermine the organ’s structural integrity and functional capacity. This condition, characterized by the progressive accumulation of scar tissue within the liver parenchyma, holds a cascade of physiological network that reverberate throughout the body, manifesting in a spectrum of symptoms and complications. From its inner onset to its profound implications for morbidity and mortality, cirrhosis generate the intricate interplay between genetic predisposition, environmental influences, and individual behaviors.

In this comprehensive discussion, we embark on an exploration of the multifaceted dimensions of cirrhosis, traversing its etiological underpinnings, clinical manifestations, diagnostic modalities, therapeutic strategies, and avenues for prevention and self-care. By delving into the intricacies of cirrhosis, we endeavor to elucidate the imperative of early recognition, proactive intervention, and holistic management in ameliorating its adverse sequelae and fostering optimal outcomes for affected individuals

Pathophysiology of Cirrhosis

Central to the pathogenesis of cirrhosis lies a complex interplay of molecular and cellular events that precipitate the gradual transformation of healthy hepatic tissue into fibrotic scar tissue. At the heart of this process lies fibrogenesis, wherein a multitude of cellular players, including hepatic stellate cells, inflammatory mediators, and extracellular matrix proteins, triggers a profibrotic milieu in response to persistent liver injury and inflammation. The ensuing deposition of collagen-rich scar tissue disrupts the structure of the liver lobules, impeding normal hepatocellular function and vascular dynamics.

Symptoms and Clinical Manifestations

The clinical presentation of cirrhosis spans a broad spectrum, reflecting the diverse array of physiological derangements engendered by advanced liver disease. Early-stage cirrhosis may manifest subtly, with nonspecific symptoms such as fatigue, malaise, and vague abdominal discomfort, often eluding timely diagnosis. However, as the disease progresses, overt signs of hepatic decompensation emerge, including jaundice, ascites, hepatic encephalopathy, and gastrointestinal bleeding, heralding the onset of life-threatening complications. Moreover, cirrhosis exerts systemic effects beyond the confines of the liver, precipitating coagulopathy, renal dysfunction, and metabolic disturbances.

Etiology and Risk Factors

Cirrhosis arises from an influence of etiological insults, encompassing a diverse spectrum of predisposing factors that contribute to hepatocellular injury and fibrogenesis. Foremost among these is chronic alcohol abuse, which inflicts direct hepatotoxic effects and potentiates oxidative stress and inflammatory cascades within the liver microenvironment. Additionally, viral hepatitis infections, particularly hepatitis B and C, constitute major drivers of cirrhosis worldwide, underscoring the imperative of vaccination and antiviral therapy in disease prevention and management. Other contributing factors include nonalcoholic fatty liver disease, autoimmune disorders, hereditary hemochromatosis, and biliary tract diseases, each exerting a unique influence on cirrhosis pathogenesis.

Diagnostic Modalities

Accurate diagnosis forms the linchpin of effective cirrhosis management, necessitating a judicious amalgamation of clinical symptoms, laboratory investigations, imaging modalities, and histopathological evaluation. Liver function tests serve as indispensable tools for gauging the synthetic and metabolic capacity of the liver, while imaging studies such as ultrasound, computed tomography (CT), and magnetic resonance imaging (MRI) afford insights into hepatic morphology, vascular dynamics, and the presence of focal lesions. Despite their utility, these modalities may be supplemented by liver biopsy, which remains the gold standard for assessing the degree of fibrosis and guiding therapeutic decisions, albeit fraught with procedural risks and sampling variability.

Treatment Strategies 

While cirrhosis represents a chronic and irreversible condition, therapeutic interventions are geared toward monitoring disease progression, alleviating symptoms, and forestalling the onset of complications. Pharmacological therapies, including hepatoprotective agents, antifibrotic agents, and symptom-based medications, aim to attenuate hepatic inflammation, inhibit fibrogenesis, and ameliorate symptom burden in cirrhotic patients. Furthermore, endoscopic interventions such as variceal band ligation and trans jugular intrahepatic portosystemic shunting play a major role in the management of portal hypertension and its sequelae, including variceal bleeding and refractory ascites. For select individuals with end-stage liver disease, liver transplantation offers a definitive therapeutic option, affording a second lease on life and circumventing the inexorable progression of cirrhosis-related complications.

Self-Care and Lifestyle

Empowering individuals with cirrhosis to adopt proactive self-care measures constitutes a cornerstone of comprehensive disease management, emphasizing the role of lifestyle modifications, dietary interventions, and preventive strategies in optimizing health outcomes. Lifestyle modifications, including abstinence from alcohol, adherence to a liver-friendly diet, regular exercise, and smoking cessation, are paramount in attenuating hepatic inflammation, promoting metabolic homeostasis, and mitigating cardiovascular risk factors in cirrhotic patients. Additionally, vaccination against hepatitis viruses, avoidance of hepatotoxic medications, and judicious monitoring of comorbid conditions serve as essential pillars of preventive care, safeguarding against disease exacerbations and fostering long-term wellness in individuals with cirrhosis.

Conclusion

Cirrhosis emerges as a complex and a dangerous disease, syndrome characterized by progressive hepatic fibrosis, compromised organ function, and a plethora of systemic manifestations. The etiological heterogeneity of cirrhosis underscores the need for a personalized and multidisciplinary approach to disease management, Focusing to the unique pathophysiological mechanisms and clinical manifestations inherent to each individual. Through timely diagnosis, targeted interventions, and collaborative care, healthcare providers can mitigate the burden of cirrhosis, optimize patient quality of life, and prolong survival in affected individuals. As we navigate complexities of cirrhosis, a concerted emphasis on preventive measures, patient education, and therapeutic innovations holds the key to unlocking a brighter prognosis for those afflicted by this formidable liver disease.

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H. pylori Infection

Introduction

Helicobacter pylori (H. pylori) infection is a significant global health concern, affecting a substantial portion of the world’s population. The discussion aims to provide an in-depth exploration of various aspects of H. pylori infection, including its prevalence, transmission, symptoms, diagnosis, treatment, complications, prevention strategies, and future research directions.

Prevalence and Transmission

H. pylori infection is widespread, with approximately two-thirds of the world’s population harboring the bacterium in their gastrointestinal tract. Various factors contribute to its prevalence, including socioeconomic status, living conditions, hygiene practices, and geographic location. The discussion indulges into the epidemiological trends of H. pylori infection across different populations and regions, highlighting disparities in prevalence rates and associated risk factors.

Transmission of H. pylori occurs primarily through interpersonal contact and ingestion of contaminated food or water. Saliva, fecal-oral transmission, and oral-oral transmission, including through kissing, are significant modes of spread. Poor sanitation and overcrowded living conditions facilitate the transmission of the bacterium, particularly in resource-limited settings. The discussion explores the mechanisms of H. pylori transmission and the implications for public health interventions aimed at reducing its spread.

Symptoms and Diagnosis

While many individuals with H. pylori infection remain asymptomatic, others experience a range of gastrointestinal symptoms, including stomach pain, bloating, nausea, and weight loss. The discussion elucidates the spectrum of clinical manifestations associated with H. pylori infection, emphasizing the importance of recognizing atypical presentations and considering differential diagnoses.

Diagnosing H. pylori infection presents several challenges due to the variability of symptoms and the limitations of available diagnostic tests. We critically evaluates the utility of different diagnostic modalities, including stool antigen tests, urea breath tests, and upper gastrointestinal endoscopy, in detecting H. pylori infection. It also examines the role of serological tests and molecular techniques in enhancing diagnostic accuracy and guiding clinical management decisions.

Treatment Options

The standard treatment regimens for H. pylori infection typically involve a combination of antibiotics and proton pump inhibitors (PPIs). However, rising rates of antibiotic resistance pose significant challenges to effective eradication therapy. It explores the mechanisms of antibiotic resistance in H. pylori and the implications for treatment outcomes.

Alternative treatment approaches, such as sequential therapy, concomitant therapy, and bismuth-based quadruple therapy, are also examined in the context of their efficacy and tolerability. Highlighting the importance of individualizing treatment regimens based on antibiotic susceptibility testing and patient-specific factors to optimize therapeutic outcomes.

Complications

Peptic ulcers are a common complication of H. pylori infection, resulting from the bacterium’s ability to disrupt the gastric mucosal barrier and induce inflammation. The discussion elucidates the pathophysiology of peptic ulcer formation and the factors contributing to ulcer recurrence and complications.

In addition to peptic ulcers, H. pylori infection is associated with an increased risk of more serious complications, such as gastric cancer and mucosa-associated lymphoid tissue (MALT) lymphoma. The discussion explores the molecular mechanisms underlying H. pylori-induced carcinogenesis and the strategies for early detection and management of gastric neoplasms.

Prevention

Preventive measures play a crucial role in reducing the burden of H. pylori infection and its associated complications. The discussion emphasizes the importance of promoting good hygiene practices, including handwashing and sanitation, to minimize the risk of transmission.

Furthermore, dietary factors may influence the risk of H. pylori infection and its clinical outcomes. The discussion evaluates the evidence regarding the impact of dietary habits, such as consumption of fruits, vegetables, and probiotics, on H. pylori colonization and disease progression. It also addresses the potential role of vaccination in preventing H. pylori infection and its complications, highlighting ongoing research efforts in vaccine development.

Research and Future Directions

Ongoing research efforts are focused on advancing our understanding of H. pylori pathogenesis, identifying novel therapeutic targets, and developing effective preventive strategies. The discussion highlights recent advancements in H. pylori research, including insights into bacterial virulence factors, host immune responses, and microbial interactions within the gastric microbiota.

Future directions in H. pylori research encompass a multidisciplinary approach, integrating molecular biology, epidemiology, immunology, and clinical medicine. The discussion outlines key areas for future investigation, such as the development of targeted antimicrobial agents, the role of host genetics in H. pylori susceptibility, and the impact of microbial dysbiosis on disease outcomes.

Conclusion

In conclusion, H. pylori infection remains a significant public health challenge, with implications for gastrointestinal health and disease worldwide. A comprehensive understanding of the epidemiology, pathogenesis, diagnosis, treatment, and prevention of H. pylori infection is essential for guiding clinical practice and informing public health policies. By addressing the complexities of H. pylori infection through interdisciplinary research and collaborative efforts, we can strive towards reducing its global burden and improving patient outcomes.

In managing H. pylori infection, compassion, empathy, and a holistic approach are crucial alongside clinical expertise. Striving for excellence in knowledge and practice enables us to advance gastroenterology and improve patient outcomes.

As we address H. pylori infection and its broader implications on gastrointestinal health, let’s remain dedicated to providing optimal patient care. By working collaboratively and embracing interdisciplinary approaches, we can positively impact lives and contribute to a healthier future.

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So I’ve been in and out of hospitals as my condition is getting bad to worse

I am a 29 years old woman living in Hungary, possibly the worst political and economically set back EU country. Our healthcare among with many other things like public education, wellfare or transport is crumbling under Orbán’s shitty policies 

This year in February I was diagnosed with MNGIE (Mitochondrial neurogastrointestinal encephalopathy) a condition that affects several parts of the body, particularly the digestive system and nervous system. Currently I am entirely deaf, underweight and in chronic pain. I am doing CAPD at home (peritoneal dialysis) and on the liver transplant list. . 

Yet my condition is worsening as I am suffering from gastrointestinal pain, vomiting and hypokalaemia. 

My doctors pretty much gave up on me. Theyre not treating my pain or my surfacing symptons or even expalining it to me. I have reached out to several clinics (one in Germany and one in the UK), but so far I haven’t even got a reply. I am without any help in this and I feel like its consuming me. 

I am without a job and my monthly income is around 80k HUF = 195 EUR = 195 USD. With current horrible inflation, that is not enough to last me through meds, the dialysis attachements, monthly trips to Budapest and my special diet. 

Please donate to my paypal if you can  paypal.me/gameofstyle

Reblogs help as well

Thank you so much!!!

/some pictures after the cut

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